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The mode of cisplatin-induced cell death in CYP2E1-overexpressing HepG2 cells: modulation by ERK, ROS, glutathione, and thioredoxin

Research output: Contribution to journalArticlepeer-review

Original languageAmerican English
JournalPharmacology, Physiology and Toxicology
StatePublished - Oct 1 2007

Keywords

  • Cisplatin
  • Extracellular signal-regulated kinases
  • CYP2E1
  • Necrosis
  • Apoptosis
  • Glutathione
  • l
  • -Buthionine-[R
  • S]-sulfoximine
  • Thioredoxin
  • Free radicals

Disciplines

  • Medical Pharmacology
  • Medical Sciences
  • Medicine and Health Sciences

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