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CYP2E1 potentiation of LPS and TNFα-induced hepatotoxicity by mechanisms involving enhanced oxidative and nitrosative stress, activation of MAP kinases, and mitochondrial dysfunction

Research output: Contribution to journalArticlepeer-review

Original languageAmerican English
JournalPharmacology, Physiology and Toxicology
StatePublished - Jun 1 2010

Keywords

  • Alcohol
  • CYP2E1
  • Lipopolysaccharide
  • Oxidative stress
  • Tumor necrosis factor alpha

Disciplines

  • Medical Pharmacology
  • Medical Sciences
  • Medicine and Health Sciences

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